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Combating Neurodegenerative Diseases with the Plant Alkaloid Berberine: Molecular Mechanisms and Therapeutic Potential

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Whilst this is true of all cell types, neurons are particularly susceptible to redox dysregulation due to their large size and high consumption of oxygen

Since up-regulation of SCN3A has been demonstrated to enhance neuronal excitability and contribute to the development of epilepsy, the reduction of SCN3A expression by VPA due to its promotion on methylation may underlie the anti-epileptic effect of VPA

Funding Open access funding provided by Chalmers University of Technology